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The fact that CRABP2 decreases in human endometrium at the time of embryo implantation might suggest that RA signaling is required to be silenced, since it shuttles RA to the RA receptors in the cell nucleus [ 7478 ]. In the mouse uterus, CRABP2 decreases around the time of embryo implantation [ 79 ] whereas P 4 induces the expression of cyp26a1, the enzyme responsible for Lallegra brigata pizzeria centocelle catabolism in mouse uterine epithelial cells [ 8081 ]. Knock down of cyp26a1 in mouse uterus decreases embryo implantation rate [ 82 ]. In addition, in human secretory endometrium, cyp26a1 mRNA level is. 20 times higher than in the proliferative phase [ 83 ]. Since the action of RA is essential for endometrial stromal cell decidualization [ 79 ] silencing of RA signaling during the window of implantation might prevent precocious decidualization of stromal cells that could compromise endometrial receptivity. The cytokine endothelin-3 (EDN3) and fibroblast growth factor receptor-1 (FGFR1) were among the transcripts consistently down-regulated in the endometrium during the window of implantation. There is abundant evidence showing that both endometrial receptivity allegra d 24 hour over the counter blastocyst implantation are regulated by cytokines and growth factors [ 84 ]. Immunoreactive pro-endothelin-3 has been described in human endometrium in luminal and glandular epithelia; however cycle-dependent regulation of this molecule is not clear [ 85 ].

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